E-Cigarette vapors have one third the amount of nicotine. E-Cigarettes develop dependency no different than tobacco.
A whole new vocabulary for parents and educators. We have seen similar trends with caffeine as well. There are hyper-caffeinated beverages today that would never have been on the market 15 years ago.
February 3, 2016
By Consumer Reports
A whole new vocabulary for parents and educators. We have seen similar trends with caffeine as well. There are hyper-caffeinated beverages today that would never have been on the market 15 years ago.
February 3, 2016
By Consumer Reports
Teens who vape—use an e-cigarette (click here) —were almost three times more likely to have started smoking tobacco cigarettes a year later than those who had never vaped, according to a recent study in the journal Tobacco Control.
“That’s worrying news,” says Consumer Reports chief medical adviser Marvin M. Lipman, M.D. “Although the long-term safety of e-cigarettes and other nicotine vaporizers is still unknown, there’s no question that nicotine in any form is an extremely addictive drug and that tobacco products can be deadly,” he says.
To find out how vaping affected teen’s cigarette smoking habits, researchers from the University of Hawaii Cancer Center asked 2,338 9th and 10th graders at six Hawaiian high schools in 2013 about their vaping and smoking habits. One year later the scientists quizzed the teens again. The upshot: 20 percent of the e-cigarette-using teens had started smoking traditional cigarettes. By contrast, 2 percent of the non-vapers had started smoking tobacco cigarettes, 10 percent had tried vaping, and 4 percent were both smoking and vaping....
The conversation among those seeking safer lives for teens talk about e-cigarettes as a gateway drug to tobacco.
The compromise of lung immunity is not unique to teens only.
February 16, 2016
By Yvette Brazier
As evidence emerges that e-cigarettes (click here) are not as safe as advertisers claim, a new study shows that flavorings classed as "Generally Recognized as Safe" by the US Food and Drug Administration are best avoided in smoking. The findings are presented at the American Association for the Advancement of Science annual meeting in Washington, DC...
...Jaspers, who is also deputy director of the UNC Center for Environmental Medicine, Asthma and Lung Biology, explains:
"The digestive systems and respiratory systems are very different. Our stomachs are full of acids and enzymes that break down food and deal with chemicals; this environment is very different than our respiratory systems. We simply don't know what effects, if any, e-cigarettes have on our lungs."
Researchers studied the effects on smokers of cinnamon-flavored e-liquids and cinnamaldehyde, the chemical that gives cinnamon flavor to an e-cigarette.
Results showed that the cinnamaldehyde e-liquids had a significant negative impact on epithelial cells that could set off a chain of cellular mechanisms potentially leading to impaired immune responses in the lung.
Jaspers elaborates: "The chemicals compromise the immune function of key respiratory immune cells, such as macrophages, natural killer cells and neutrophils."...
Lung physiology
Innate immunity (click here) is a primordial system that has a primary role in lung antimicrobial defenses. Recent advances in understanding the recognition systems by which cells of the innate immune system recognize and respond to microbial products have revolutionized the understanding of host defenses in the lungs and other tissues. The innate immune system includes lung leukocytes and also epithelial cells lining the alveolar surface and the conducting airways. The innate immune system drives adaptive immunity in the lungs and has important interactions with other systems, including apoptosis pathways and signaling pathways induced by mechanical stretch. Human diversity in innate immune responses could explain some of the variability seen in the responses of patients to bacterial, fungal, and viral infections in the lungs. New strategies to modify innate immune responses could be useful in limiting the adverse consequences of some inflammatory reactions in the lungs...
The conversation among those seeking safer lives for teens talk about e-cigarettes as a gateway drug to tobacco.
The compromise of lung immunity is not unique to teens only.
February 16, 2016
By Yvette Brazier
As evidence emerges that e-cigarettes (click here) are not as safe as advertisers claim, a new study shows that flavorings classed as "Generally Recognized as Safe" by the US Food and Drug Administration are best avoided in smoking. The findings are presented at the American Association for the Advancement of Science annual meeting in Washington, DC...
...Jaspers, who is also deputy director of the UNC Center for Environmental Medicine, Asthma and Lung Biology, explains:
"The digestive systems and respiratory systems are very different. Our stomachs are full of acids and enzymes that break down food and deal with chemicals; this environment is very different than our respiratory systems. We simply don't know what effects, if any, e-cigarettes have on our lungs."
Researchers studied the effects on smokers of cinnamon-flavored e-liquids and cinnamaldehyde, the chemical that gives cinnamon flavor to an e-cigarette.
Results showed that the cinnamaldehyde e-liquids had a significant negative impact on epithelial cells that could set off a chain of cellular mechanisms potentially leading to impaired immune responses in the lung.
Jaspers elaborates: "The chemicals compromise the immune function of key respiratory immune cells, such as macrophages, natural killer cells and neutrophils."...
Lung physiology
Innate immunity (click here) is a primordial system that has a primary role in lung antimicrobial defenses. Recent advances in understanding the recognition systems by which cells of the innate immune system recognize and respond to microbial products have revolutionized the understanding of host defenses in the lungs and other tissues. The innate immune system includes lung leukocytes and also epithelial cells lining the alveolar surface and the conducting airways. The innate immune system drives adaptive immunity in the lungs and has important interactions with other systems, including apoptosis pathways and signaling pathways induced by mechanical stretch. Human diversity in innate immune responses could explain some of the variability seen in the responses of patients to bacterial, fungal, and viral infections in the lungs. New strategies to modify innate immune responses could be useful in limiting the adverse consequences of some inflammatory reactions in the lungs...
