Clots most likely triggered by cytolysis.

...As Oxley, (click here) an interventional neurologist, began the procedure to remove the clot, he observed something he had never seen before. On the monitors, the brain typically shows up as a tangle of black squiggles — “like a can of spaghetti,” he said — that provide a map of blood vessels. A clot shows up as a blank spot. As he used a needlelike device to pull out the clot, he saw new clots forming in real-time around it....

Besides this phenomenon, there is another entry in my reading that mentions the discovery of lung tissue that was a mass of tissue without any real function. That is more than likely cytolysis used by the virus to release the replicated virus from the cell.

Where is the vaccine that will end the ability for the virus to attach to healthy human cells, using them and destroying it's plasma membrane.

The virus is not only triggering inflammation in the immune response it is lysing cells that release new virus. The lysing is triggering the clotting cascade.

That is my taking on it.

Critical care of these patients may at time include supporting the patient with elements that provide for an intact clotting cascade. Running out of the elements of the clotting cascade will result in DIC (disseminated intravascular coagulation).

Basically, the SARS-CoV-2 RNA attaches to the ACE2 (angiotensin I converting enzyme 2 [ Homo sapiens (human) ] (click here))
site of the human cell to enter and deposit the RNA into the cell, the RNA uses the cell's DNA resulting in the replicated virus. The replicated virus has to get out of the cell to continue the infection process, it destroys the human cell plasma membrane, hence, destroying the cell. The immune reaction of the human body causes inflammation and clotting to address cell destruction. Cell destruction (lysis) releases chemical components that alert the body's immune response and blood clotting.

...We show that SARS-CoV-2 S uses ACE2 (click here) to enter cells and that the receptor-binding domains of SARS-CoV-2 S and SARS-CoV S bind with similar affinities to human ACE2, correlating with the efficient spread of SARS-CoV-2 among humans....SARS-CoV-2 S glycoprotein harbors a furin cleavage site at the boundary between the S1/S2 subunits...murine polyclonal antibodies potently inhibited SARS-CoV-2 S mediated entry into cells...

Until the vaccine:

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